Please use this identifier to cite or link to this item: http://hdl.handle.net/123456789/5305
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dc.contributor.authorCompaore, Tegwinde Rebeca-
dc.contributor.authorSoubeiga, Serge Theophile-
dc.contributor.authorAbdoul Karim Ouattara-
dc.date.accessioned2021-04-08T11:59:57Z-
dc.date.available2021-04-08T11:59:57Z-
dc.date.issued2018-06-
dc.identifier.urihttp://hdl.handle.net/123456789/5305-
dc.description.abstractAbstract APOBEC3G is a potent inhibitor of HIV-1 replication, and act by deaminating cytidines in uracil on the negative strand of the viral cDNA. In this case-control study, APOBEC3G expression in subjects’ naïve to HAART infected by HIV-1 and the effect of APOBEC3G polymorphism on its expression were evaluated. The results show that the HIV-1 infected carriers of the G minor alleles of the variant rs8177832 had a higher expression of APOBEC3G mRNA than the controls carriers of the G minor allele. APOBEC3G polymorphisms could play an important role in the modulation of the HIV-1.en_US
dc.language.isoenen_US
dc.subjectAPOBEC3Gen_US
dc.subjectHIV-1en_US
dc.titleAPOBEC3G expression and HIV-1 infection in Burkina Fasoen_US
dc.typeArticleen_US
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